COURT OF APPEALS DECISION DATED AND RELEASED JUNE 20, 1995 |
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No. 94-3190
STATE
OF WISCONSIN IN COURT OF
APPEALS
DISTRICT III
ORVILLE H. WERNER,
Plaintiff-Appellant,
v.
LABOR AND INDUSTRY
REVIEW
COMMISSION, EDGAR
PACKING
COMPANY, INC. and
TRANSPORT
INSURANCE COMPANY,
Defendants-Respondents.
APPEAL from a judgment
of the circuit court for Marathon County:
RAYMOND F. THUMS, Judge. Affirm.
Before Cane, P.J.,
LaRocque and Myse, JJ.
LaROCQUE, J. Orville Werner appeals a judgment affirming
a decision of the Labor and Industry Review Commission. The commission dismissed Werner's
application for worker's compensation, finding that Werner's exposure to
ammonia vapor in the work place was not proven to cause his interstitial lung
disease. LIRC based its decision on the
medical reports of Dr. David Jolin, finding them more persuasive than the
testimony of Werner's treating physician, Vinoo Cameron, who concluded that
Werner's lung disease was caused by exposure to ammonia. Werner argues that LIRC's decision is not
supported by credible and substantial evidence in the record. We reject Werner's argument and affirm the
trial court order upholding LIRC's decision.
BACKGROUND
Orville Werner has
interstitial lung disease. Werner
worked for seventeen years for the Edgar Packing Company until he retired in
1985. For sixteen of those years, he
worked in a refrigerated cooler that had a chronic ammonia leak, resulting in
his exposure to an undetermined amount of anhydrous ammonia vapor during the
course of his employment.[1]
Two hearings were held
before a Department of Industry, Labor and Human Relations administrative law
judge, who found Werner to be permanently and totally disabled and ordered
Edgar Packing and its compensation insurer to pay worker's compensation
benefits to Werner. Edgar Packing and
the insurer petitioned for review. LIRC
made its own findings of fact and reversed the order, concluding that it would
be required to speculate to find that Werner's interstitial lung disease arose
out of and in the course of his employment. Werner appealed to the circuit
court, which affirmed LIRC. Werner
argues that the record contains insufficient evidence to support Jolin's opinion
that Werner did not have a chronic, productive cough or symptoms of
bronchiectasis, which he stated would be the usual sequelae of chronic ammonia
exposure.
DISCUSSION
This court reviews the
LIRC decision. West Bend Co. v.
LIRC, 149 Wis.2d 110, 117, 438 N.W.2d 823, 827 (1989). Our scope of review is identical to that of
the circuit court. Oscar Mayer
Foods Corp. v. LIRC, 145 Wis.2d 864, 868, 429 N.W.2d 89, 91 (Ct. App.
1988). Resolution of the question
whether a medical condition arose out of an applicant's employment may require
LIRC to choose between conflicting medical testimony. See id.
It is not the function of this court to weigh disputed medical
testimony; the commission's finding on disputed medical testimony is
conclusive. Worsch v. DILHR,
46 Wis.2d 504, 512, 175 N.W.2d 201, 206 (1970). Moreover, LIRC's findings of fact are conclusive if there is any
credible evidence to support them. West
Bend Co., 149 Wis.2d at 117-18, 438 N.W.2d at 827. The test for credible evidence is whether
the evidence is relevant, evidentiary in nature and not a conclusion of law,
and not so completely discredited by other evidence that a court could find it
incredible as a matter of law. Worsch,
46 Wis.2d at 513, 175 N.W.2d at 206.
The question is not whether there is credible evidence in the record to
sustain a finding the commission did not make, but whether there is any
credible evidence to sustain the finding that the commission did make. Mednicoff v. DILHR, 54 Wis.2d
7, 18, 194 N.W.2d 670, 675-76 (1972).
We conclude that substantial and credible evidence supports LIRC's
finding of legitimate doubt that Werner's condition arose out of or was
incidental to his employment. LIRC
could reasonably conclude that Werner failed to meet his burden of proof
because it could only speculate concerning the cause of his condition.
In this case, Cameron
testified that in his opinion, exposure to ammonia gas burned Werner's lung
tissue, causing scarring and fibrosis.
He stated, "I have no other explanations. ... The kind of fibrosis he has is either from a gas that went
into the lungs or some other poison that went in. It's not normal." He
continued, "it is plausible in my medical judgment that that ammonia gas would
have caused [Werner's lung disease] because I don't know what else would."
Jolin, an internist who
examined Werner at the request of Edgar Packing, stated that there is no
scientific evidence that long-term chronic exposure to ammonia fumes can cause
interstitial lung disease. He stated
that interstitial lung disease can be caused by inhaling gases such as chlorine
or sulfur dioxide; inhalation of organic dusts, including aspergillus mold;
drug reaction; infectious agents such as viruses; or for no readily discernable
reason. Jolin gave the opinion that, to
a reasonable degree of medical certainty, Werner's lung disease was idiopathic,
i.e., of unknown origin, or was due to aspergillus mold, a recognized
cause of interstitial lung disease.[2] Jolin concluded that "[i]t would be
pure speculation to suggest, contrary to the scientific literature, that
ammonia is the cause of his interstitial lung disease."
Werner
contends that Jolin's opinions, upon which LIRC's findings are based, are not supported
by credible evidence in the record.
First, Werner claims that although Jolin based his opinion in part on
Werner's lack of a productive cough, witnesses testified that Werner exhibited
a heavy cough when working in the cooler.
Second, he asserts that although Jolin found no evidence of chronic
bronchiectasis, there was x-ray evidence of bronchiectasis. Third, he argues that LIRC should not base
its decision on Jolin's opinion that "[t]here is no scientific evidence
that long-term chronic exposure to ammonia fumes can produce interstitial lung
disease" because, he suggests, there are such studies, and also because
LIRC's decision results in his being punished for the absence of such studies.
Cameron testified that
when he first examined Werner in 1988, Werner was short of breath and his lungs
were damaged. In his written report
dated November 11, 1988, Cameron noted Werner reported an "[o]ccasional
history of cough." Cameron did not
report a chronic cough. Neither did
Jolin. In his examination of Werner,
Jolin reported that "[a]uscultation of the lungs revealed mild to
moderately diminished breath sounds ... and a few fine crackles were heard
...." Jolin's report recounts that
Werner reported an "occasional cough which is usually non-productive,
though he does report production of 'brownish-yellow' sputum on infrequent
occasions." No evidence in the
record suggests that Werner had the chronic, productive cough that Jolin
reported is a characteristic symptom of chronic ammonia exposure.[3]
In his supplemental
report, Jolin addressed the issue of Werner's cough, noting that witnesses had
testified "that Mr. Werner was observed coughing on numerous occasions at
work." However, Jolin said,
"[coughing] is a nonspecific symptom and cannot specifically be related to
ammonia exposure. Coughing is an upper
respiratory symptom and does not indicate that lower respiratory injury is
occurring." He suggested other
possible causes: "Mr. Werner's
past history of smoking, exertion due to his employment, colds, or the cold,
dry air of the freezer in which he worked."
Werner also contends
that an x-ray dated December 30, 1991, showed evidence of bronchiectasis. Cameron interpreted this x-ray as
"suggesting an element of possible bronchiectasis." Jolin also addressed the issue of
bronchiectasis in his supplemental report, stating, "Mr. Werner is
alleged to have bronchiectasis on one x-ray.
The x‑ray taken at the time of my examination did not disclose any
bronchiectasis. Further, as I commented
in my initial report, the absence of a productive cough tends to indicate there
is no bronchiectasis. I believe the
report of bronchiectasis on a previous x-ray is incorrect."
In reaching its
decision, LIRC noted that Jolin's extensive research in the applicable medical
literature revealed no evidence that chronic exposure to ammonia could cause
interstitial lung disease, and that reports of the perception of ammonia odor
do not establish that ammonia was present in sufficient concentration to have
caused respiratory injury. LIRC also
noted Jolin's statement that there are many possible causes for interstitial
lung disease, one of which is exposure to aspergillus mold, for which Werner
tested positive.
LIRC
noted further that Cameron's opinion was based on studies involving a small
number of patients who had been the victims of one acute exposure to ammonia
rather than prolonged exposure, as in Werner's case. Jolin stated:
In particular, the Close Report, Exhibit
9, dealt with persons who had experienced "prolonged exposure,"
defined by them as being more than one-half hour. It dealt with only six patients in that category. All had chemical burns of one sort or
another from the ammonia, and all had full thickness airway burns. There is no evidence of such burns in Mr.
Werner's examination.
In
summary, the alleged exposure of Mr. Werner is different in nature than the
acute exposure in the studies and they have no application to his case.
LIRC accepted Jolin's
opinion that the clinical studies submitted into evidence by Cameron did not
support the factual inference that chronic exposure to low levels of ammonia
gas could cause interstitial fibrosis of the lungs. LIRC noted that there was no clinical data to support the
postulation in one study that chronic exposure to anhydrous ammonia vapor in
low concentrations probably would result in extensive alkali burns of the
tracheobronchial tree. LIRC concluded,
based on its review of the evidence and consultation with the administrative
law judge, that it was left with a legitimate doubt that the applicant's work
exposure to ammonia vapor was the cause of his lung disease.
Werner suggests research
has been done on chronic exposure to ammonia, research that was not available
to counsel or doctors at the original hearings. However, if Werner had evidence he might have presented but
failed to present, he cannot now collaterally attack the decision by claiming
that he had affirmative evidence in the action that he did not use. See Conway v. DNR, 50
Wis.2d 152, 160, 183 N.W.2d 77, 81 (1971).
Werner argues that
"there is nothing in the literature that suggests that it is impossible
for repeated exposures to cause damage to the lungs." However, the burden was on Werner to produce
sufficient evidence to remove the question in dispute from the realm of speculation. See Franckowiak v. LIRC, 12
Wis.2d 85, 87-90, 106 N.W.2d 51, 52-53 (1960).
The employer was not required to prove that Werner's lung disease was
caused by factors unrelated to his work.
See id. at 88, 106 N.W.2d at 52. Werner is not being punished for the absence of scientific
studies. The evidence is in conflict,
which is not a sufficient basis for the reversal of LIRC's findings. Eastex Packaging Co. v. DILHR,
89 Wis.2d 739, 745, 279 N.W.2d 248, 250 (1979).
We have reviewed and
considered the record and each of the points Werner raised. We are of the opinion that his arguments do
not demonstrate Jolin's medical testimony to be incredible, but attack the
weight given by LIRC to the evidence.
The commission consulted
with the administrative law judge and set forth the reasons for its contrary
findings of ultimate fact. See Goranson
v. DILHR, 94 Wis.2d 537, 546, 289 N.W.2d 270, 275 (1980) (LIRC required
to make findings of ultimate facts as distinguished from evidentiary
facts). LIRC is not required to make a
finding that it finds the testimony of any witness incredible. Bowen v. Industrial Comm'n,
239 Wis. 306, 312, 1 N.W. 77, 80 (1941).
LIRC concluded that Jolin's opinion was more persuasive than Cameron's
and resolved the issue of causation against Werner on the basis of crediblility
of medical evidence. Jolin's medical
opinion was not discredited by other evidence so that this court could find it
incredible as a matter of law.
Therefore, LIRC's findings are affirmed.
By the Court.—Judgment
affirmed.
Not recommended for
publication in the official reports.
[1] Major leaks occurred one to two times per month, with a strong presence of ammonia 12 to 18 times per year. The amount of ammonia vapor to which Werner was exposed was never measured, and there is no evidence in the record indicating how long each individual exposure lasted.
[3] Werner was hospitalized in February 1988. In a consultation record from Wausau Hospital dated February 9, 1988, the consulting physician, Dr. Rick Reding, noted that Werner exhibited a persistent, nonproductive cough and that for 32 years Werner had smoked one and one-half packs of cigarettes per day and had stopped 10 years earlier. In a follow-up record dated February 29, 1988, Reding noted, "[h]e denies any cough."